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Tag: heart failure

CRC1425: PhD and PostDoc positions available

Posted on2020-06-032023-03-13

The newly established German Collaborative Research Centre (SFB1425) “Heterocellular Nature of Cardiac Lesions: Identities, Interactions, Implications” is recruiting 13 PhD students and 5 post-docs to join cutting-edge research that aims to develop insight and tools to improve clinical outcomes after more…

CategoriesNewsTagscardiac myocytes, endothelial cells, fibroblasts, heart, heart failure, SFB1425

Otto-Hess Award 2019

Successful Otto Hess scholarship

Posted on2019-10-152019-11-25

Jessica Kowalski, MD student and Cardiovascular Pharmacology Research Group member, successfully completed her Otto Hess Scholarship, funded by the German Cardiac Society. For her work on the cell type-specific role of the mineralocorticoid receptor in pulmonary hypertension and right heart more…

CategoriesNewsTagsDGK, heart failure, mineralocorticoid receptor, Pulmonary hypertension

Cytokine adsorption in patients on ECMO

Posted on2019-05-252019-05-25

In this prospective single-center registry we evaluated the use of cytokine adsorption in critically ill patients requiring extracorporeal membrane oxygenation (ECMO) support. Sepsis but also in non-infectious conditions such as post-cardiac arrest syndrome are associated with severe systemic inflammation. Cytokine more…

CategoriesNews, PublicationsTagscardiac arrest, ECMO, heart failure, intensive care

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aldosterone atrial fibrillation biomarker cardiac arrest cardiac myocytes COVID-19 DGK diabetes ECMO endothelial cells epigenetics fibroblasts gene expression heart heart failure heart transplant hypertension inflammation intensive care kidney mineralocorticoid receptor Pulmonary hypertension SFB1425 transcription factor vascular smooth muscle cells

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  • Downregulation of TCF19 and ATAD2 causes endothelial cell cycle arrest at the transition from cardiac hypertrophy to heart failure
  • Cell Type–Resolved Transcriptome of Aldosterone-Induced Atrial Myopathy and Arrhythmia
  • Gene expression networks in endothelial cells from failing human hearts

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